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Thus, H 2 S-producing enzymes may or may not become upregulated in various models of diabetes, perhaps depending on the experimental model and/or the severity of the disease (but it appears that they are certainly not down-regulated)

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NPC radioresistance is attributed to various regulatory mechanisms, including DNA damage response signalling, cancer stem cell phenotype, abnormal cell-cycle progression, hypoxic properties, and the tumour immune microenvironment [42,43,44,45]