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FIGURE 5 Ferroptosis, a regulatory cell death mode, is characterized by iron-dependent and lipid peroxide (LPO) accumulation, proposed by 2+ , which can produce •OH through the Fenton reaction, and then react with polyunsaturated fatty acids in the plasma membrane to produce ROS, resulting in iron-regulated death

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The loss of HDAC1 in neurons causes DNA damage and cell death, while the lack of HDAC2 in mice improves learning and memory, enhances synaptic plasticity, and increases dendritic spine density and synapse number (89)