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Within synaptic vesicles, amphetamine disrupts vesicular monoamine storage through two complementary mechanisms: (A) diffusion into vesicles and proton buffering, which collapses the vesicular proton gradient required for vesicular monoamine transporter 2 (VMAT2) function

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While many tumors harbor p53 loss-of-function mutations or deletions, others contain oncogenic gain-of-function mutations of p53 , resulting in the expression of mutant p53 protein in tumors, and such tumors often exhibit poor prognosis and aggressive tumor phenotypes (Muller and Vousden, 2013)